I have been advised to start taking ginkgo biloba and Ginseng, L-carnitine and Coenzyme Q10.Long term effects?
I have been advised to start taking ginkgo biloba and Ginseng, L-carnitine and Coenzyme Q10.Will there be any long term effects?
My mum takes Ginkgo biloba and has stopped for a month and her memory and brain are always muddled and forgetful.
My worry is that once you start taking these things you need to stick with them?
Surely if you start taking, your body stops producing and therefore if you don’t take them you end up with a deficiency?
Any advice would be great, many thanks
Advised by my doctor for CFS
Advised by whom and for what purpose?
Can I take 5-HTP, SAMe, tyrosine, l-carnitine and CoQ-10 together?
I have been suffering from on and off depression for most of my young adult life (i’m 19 years old at the moment), and am getting a number of possible organic causes tested for. I have had diabetes, celiac disease and nutritional deficiencies ruled out, and am next attempting to rule out allergies, mineral imbalances, toxicity, and hypto-thyroidism. If these don’t result in any breakthroughs i’m resorting to self- medicating for depression.
I plan to take SAMe and tyrosine in the morning, l carnitine and CoQ-10 a few hours later, and 5-HTP right before I go to sleep. Is there a possibility of any negative reactions among these drugs?
In almost all vitamin stores and health food stores, I have seen sections of the store devoted to “brain supplements” or “mood enhancers.” Many of the ingredients in the brain and mood products are in combination form. In other words, a product in the Vitamin Shoppe, for example, might contain the amino acids L-carnitine and L-tyrosine plus herbs like ginkgo biloba and St. John’s wort, and possibly CoQ10, phosphatidyl serine, 5-HTP, etc.
Research on St. John’s wort suggests it can be helpful for mild (or maybe moderate) depression. However, what many people don’t know is that it can take at least 3 weeks to start working. This herb can also interact with some medications. So you should ask a health care professional about any possible bad interaction if you are taking any prescription medication.
I suggest you start with St. John’s wort since it has been researched more methodically than the other products you mentioned.
The products you mentioned may or may not be helpful for you. There is not a lot of scientific proof that they work. There is only speculation and “testimonials.”
I suggest you avoid SAMe. I go to a weekly mood support group for people with depression, bipolar disorder, etc. SAMe is very expensive and I have never heard anyone report that it helped them.
Finally, I doubt there would be negative interactions in the products you mentioned. Most of them have been sold in combination forms for years.
For myself, I found exercise to be a very potent antidepressant. I also practice relaxation techniques like meditation. Psychotherapy has also been helpful for me.
biochem q on LCAD and glycogen storage diseases?
Fraternal twins have episodes of fasting hypoglycemia, hypoketonemia, and muscle weakness
occurring only during periods of caloric deprivation.
One twin dies at 5 months of age.
surviving twin develops a cardiomyopathy, which is diagnosed at 3 years of age.
are elevated in muscle tissue and in serum.
Which of the following is the most likely diagnosis?
a-L-Iduronidase deficiency hunters
Acid maltase deficiency (a 1,4-glucosidase deficiency) pompes
Carnitine uptake deficiency
Long-chain acyl CoA dehydrogenase (LCAD) deficiency
Muscle glycogen phosphorylase deficiency mcardles
i was torn between B and D on this q…since carnitine levels were high then id say D. but what else could distinguish this answer of d being correct? i said pompes aka b first since the infant died and it was a heart problem plus the symptoms were aggrevated in times of not eating. pompes is a glycogen storage disease where a lysozomal enzyme doesnt break down glyocgen so if someone didnt eat and needed the glycogen theyd be screwed with this genetic disorder. any help is appreciated.
I agree that the elevated carnitine levels point to answer D. But I am not sure of this. What mystifies me is why these children do not have elevated ketones.
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