Your Questions About An Impaired Immune System

February 2, 2014
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Daniel asks…

Is there a limit to how many dextro energy tablets you can have?

Just wondering if there is. Because I cannot find anything on the packet. Is there a danger if I have a whole pack today?

admin answers:

There is no info on the energy tablet sites, they are 89% dextrose.
Dextrose is more or less an industry term for glucose. Glucose is the most prevalent sugar in the human and the only molecule that the brain can metabolize. It has a very high glycemic index (no surprise since it is glucose) and while it contains no fructose, it is still a simple sugar that is very readily absorbed.

Nearly every cell in your body utilizes glucose, so it’s normally “burned up” immediately after consumption.
Too much sugary stuff can suppress your immune system and impair your defenses against infectious disease, lead to hyperactivity, weight gain, tooth decay etc etc.

So while there is no ‘safe’ limit to worry about use your common sense about how many you eat. If you notice they have an adverse effect on you then you know to stop eating them, like if you feel hyperactive or anxious.
As necessary would seem a good rule to apply.

David asks…

What is the difference between the Multiple Sclerosis and the Friedreich syndrom?

My brother has something that goes between both of them and even doctors couldn’t identify it.he also has a malformation of his feet bones.

admin answers:

Multiple sclerosis:

Abbreviated MS, also known as disseminated sclerosis.
Is a chronic, inflammatory disease that affects the central nervous system (CNS). MS can cause a variety of symptoms, including changes in sensation, visual problems, muscle weakness, depression, difficulties with coordination and speech, severe fatigue, and pain. Although many patients lead full and rewarding lives, MS can cause impaired mobility and disability in the more severe cases.

Multiple sclerosis affects neurons, the cells of the brain and spinal cord that carry information, create thought and perception and allow the brain to control the body. Surrounding and protecting some of these neurons is a fatty layer known as the myelin sheath, which helps neurons carry electrical signals. MS causes gradual destruction of myelin (demyelination) and transection of neuron axons in patches throughout the brain and spinal cord, causing various symptoms depending upon which signals are interrupted. The name multiple sclerosis refers to the multiple scars (or scleroses) on the myelin sheaths. It is thought that MS results from attacks by an individual’s immune system on the nervous system and is therefore categorized as an autoimmune disease.

Multiple sclerosis may take several different forms, with new symptoms occurring in discrete attacks or slowly accruing over time. Between attacks, symptoms may resolve completely, but permanent neurologic problems often persist. Although much is known about how MS causes damage, its exact cause remains unknown. MS currently does not have a cure, though several treatments are available which may slow the appearance of new symptoms. MS primarily affects adults, with an age of onset typically between 20 and 40 years, and is more common in women than in men.

Friedreich Ataxia:

Friedreich’s ataxia is an inherited disease that causes progressive damage to the nervous system resulting in symptoms ranging from gait disturbance and speech problems to heart disease. It is named after the German physician Nicholaus Friedreich, who first described the condition in the 1860s. “Ataxia,” which refers to coordination problems such as clumsy or awkward movements and unsteadiness, occurs in many different diseases and conditions. The ataxia of Friedreich’s ataxia results from the degeneration of nerve tissue in the spinal cord and of nerves that control muscle movement in the arms and legs. The spinal cord becomes thinner and nerve cells lose some of their myelin sheath the insular covering on all nerve cells that helps conduct nerve impulses.

Friedreich’s ataxia, although rare, is the most prevalent inherited ataxia, affecting about 1 in every 50,000 people in the United States. Males and females are affected equally.

Friedreich’s ataxia is an autosomal recessive congenital ataxia and is caused by a mutation in Gene X25 that codes for frataxin, located on chromosome 9. This protein is essential in neuronal and muscle cells for proper functioning mitochondria and the mutation causes inadequate production of frataxin. This results in, among other things, a degeneration of nerve tissue in the spinal cord.

Delatycki et al. (2000) provided an overview of the clinical features, pathology, molecular genetics, and possible therapeutic options in Friedreich ataxia.

Friedreich’s ataxia and muscular dystrophy, though often compared, are completely different diseases. Muscular dystrophy is the result of muscle tissue degeneration whereas Friedreich’s ataxia is the result of nervous tissue degeneration caused by a trinucleotide repeat expansion mutation. Both are researched by the Muscular Dystrophy Association.

There are two types, the classic form and one in association with a genetic vitamin E deficiency. They cannot be distinguished clinically.

Jenny asks…

What to use for rain rot and how to prevent it…?

My gelding gets rain rot every year around this time or during the winter months no matter what I do to try and prevent it. I can’t give him a bath because it’s too cold where I live, but what other type of treatment could I use to get rid of it? And how can I prevent him from getting it again? Would a coat supplement, vitamin supplement, or corn oil help? Please help we both would really appreciate it!

Thanks!

PS: He also wears a blanket in the winter.

admin answers:

The dermatophilus congolensis bacteria that cause it live dormant in the skin. When the immune system functions are compromised, or the integrity of the skin is compromised, the bacteria can begin to colonize and develop an active infection in the skin. Every time your horse has an active infection, it can be spread to a wider area or into deeper layers of tissue if you are not careful in handling it.
This is not a fungal infection which only affects dead surface epidermis and hair. This affects living tissue and is treated differently than a fungus is. The products being suggested in other answers are more appropriate for fungal infections, and not for an infection of living tissue.

Good health management is the best preventive. Deworming as needed based on fecal egg counts is one important preventive. Regular vaccinations are another. A well balanced diet with proper balance of proteins, carbohydrates, fat, minerals, and vitamins is important, and regular veterinary evaluation for any metabolic or other conditions that influence the dietary and other needs is a good preventive.

Skin is protected by what is called an “acid mantle”. The natural oils from sebaceous glands, acid pH in sweat, dead cells and bacterial microflora of the skin all work together to maintain the acid mantle and create a barrier between the environment and the skin. This barrier protects against vectors like insects and the microflora compete against pathogenic microbes to prevent infections. The hair coat also protects the skin from harmful UV light rays, and the oils maintain its waterproofing capability.

If you bathe the horse using any kind of soaps or disinfectants, you will strip the skin and hair oils and remove dead cells, alter the pH, and kill off the microflora. Obviously this all impairs skin integrity and can allow injury or infection leading to rain rot.

If you blanket your horse and he overheats and sweats beneath the blanket, the prolonged exposure to dampness against the skin can impair integrity and allow the bacteria to become active and produce rainrot. Leaving a horse out in rain and never bringing him in to get dried off has the same effect. D. Congolensis bacteria are anaerobic, so they thrive in damp, dark, airless environments. As the skin barrier to moisture breaks down from prolonged exposure, the anaerobes begin to thrive and multiply.

So, keeping your horse in overall good health, NOT bathing the coat and skin with soaps and disinfectants, drying the coat and skin out periodically during wet weather, and not leaving blankets on when a horse is sweating underneath it are all preventive. Lots of exercise and fresh air is best, and 24/7 with a run in is ideal, Take precautions against spread if another horse has an outbreak of rain rot. It is transmissible by direct contact, on fences, tack and equipment, and by vectors like flies. The healthier your horse is, and the better his skin integrity is, the less risk there is that he will be newly infected or have a recurrence.

It is also tranmissible to humans, so take precautions.

Supplements only help if they are providing what is needed. I have fed Enreco Omega Horseshine which is a flax linseed oil meal and have always had great results for horses with dry skin or dull coats and you can usually safely add that to your horse’s feed. Compared to some of the others out there, it is a better quality product. Smart pack carries it or you can buy direct online.

This is Horseshine…………………

Http://www.valleyvet.com/ct_detail.html?pgguid=ed970fcd-953e-4880-9757-783c69d02c6e

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Add……………….. If your horse does develop rain rot, you can gently soak the scabs in saline solution and when they soften and loosen, remove them with cotton pads (I use makeup removal pads). You don’t want to drive the infection deeper or spread it around, so be gentle and meticulous. Then rinse with more saline to debride the sores and pat dry. Leave the sores exposed to air which kills the anaerobic bacteria. Be sure to dispose of the scabs in a sealed container as they are loaded with bacteria. A thin layer of EMT can be applied to seal out insects, block pain and itch and promote healing. You can also use Vetericyn. Just be sure to air dry the open sores first. If the skin is swollen, the infection is probably in deeper tissue layers, and your vet can administer antibiotic injections to eradicate the infection.

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